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Fluoxetine ( Prozac ) administered at time of carrageenan injection or 30 min after carrageenan challenge, markedly inhibited the paw oedema response. The
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anti-oedema effects of Fluoxetine ( Prozac ) and melatonin or the tricyclic antidepressant imipramine were additive. Fluoxetine ( Prozac ) (360 or 720 microg per paw) given into the rat paw with carrageenan reduced the oedema response by 25.4 and 35.3% 4 h post-carrageenan, respectively.
These drugs, such
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as Fluoxetine ( Prozac ) and Paroxetine ( Paxil ), exert their antidepressant activity by increasing 5-HT concentration in the synaptic cleft and thus enhancing serotonergic neurotransmission.
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Those physically coupled to G proteins) appeared to remain unaltered all along a 3-week treatment with either Fluoxetine ( Prozac ) or Paroxetine ( Paxil ). Both the serotonergic
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and the opioid systems are likely to be involved in the modulating action of Fluoxetine ( Prozac ) on peripheral inflammation..
Coupled and uncoupled with regard to G proteins) revealed no significant change in rats treated with either SSRI. Several hypothetical mechanisms
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have been put forward to account for this delay, taking into account pharmacokinetic considerations, neurotransmitter metabolism, and/or adaptive regulation of pre and/or post-synaptic receptors. Accordingly,
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the proportion of functional 5-HT1A receptors (i.e. Comparative effects of Fluoxetine ( Prozac ) and Paroxetine ( Paxil )The hypothesis that a dysfunction of serotonergic neurotransmission is implicated in depression
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is supported by the clinical efficiency of selective serotonin drug store delivery (5-hydroxytryptamine, 5-HT) reuptake inhibitors
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(SSRIs) in the treatment of depressive disorders.
In vitro binding and quantitative autoradiographic studies sho that neither 5-HT1A, 5-HT1B, 5-HT2A, nor 5-HT3 receptor binding sites in various brain areas were affected by these treatments. Studies on the anti-inflammatory ranitidine medicine effect of Fluoxetine ( Prozac ) in the rat.The anti-inflammatory activity of Fluoxetine
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( Prozac ), a selective serotonin reuptake inhibitor (SSRI), was studied on the carrageenan-induced paw
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inflammation in the rat. The aim of this study was to look for such adaptive changes in the course of a 3-week treatment with Fluoxetine ( Prozac ) (5 mg/kg/day, i.p.) or Paroxetine ( Paxil ) (5 mg/kg/day, i.p.) in adult rats. Fluoxetine ( Prozac ) (10 or 20 mg kg(-1), i.p.) co-administered with
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indomethacin (IND) (20 mg kg(-1),
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i.p.), celecoxib (10 mg kg(-1), i.p.) or rofecoxib (4.5 mg kg(-1), i.p.) before carrageenan reduced the anti-oedema effect of indomethacin or celecoxib, but had additive effect to that of rofecoxib. The anti-oedema
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effect of Fluoxetine ( Prozac ) was partially suppressed by the opioid antagonist naloxone (4 mg kg(-1), i.p.). Fluoxetine ( Prozac ) (10-60 mg kg(-1)) given intraperitoneally (i.p.) 30 min before carrageenan, displayed marked anti-inflammatory
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activity, inhibiting paw oedema by 38.6-77.7% at 2 h post-carrageenan. The proportion of recorded neurons showing desensitization of somatodendritic 5-HT1A autoreceptors then increased along the treatment, and was generally larger with Fluoxetine ( Prozac ) than with Paroxetine
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Thus, the potency of the 5-HT1A autoreceptor agonist, 8-OH-DPAT, to depress the firing of serotonergic neurons in brain
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stem slices was significantly reduced as soon as after a 3-day treatment with either SSRI. In contrast, administration of both Fluoxetine ( Prozac ) and the heterocyclic antidepressant trazodone had no greater anti-inflammatory effect than Fluoxetine ( Prozac ) alone. liquid anastrozole Nevertheless,
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in vitro electrophysiological
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recordings of serotonergic neurons in the dorsal raphe nucleus allo the demonstration of a clearcut functional desensitization of somatodendritic 5-HT1A autoreceptors. As 5-HT1A autoreceptor desensitization can contribute to facilitate serotoninergic neurotransmission, the remarkable efficiency of Fluoxetine ( Prozac ) to trigger this adaptive regulatory mechanism might account, at least partly,
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for its potent antidepressant activity. Furthermore, comparison of the specific binding of 8-OH-DPAT to 5-HT1A receptors functionally coupled to G proteins with that of WAY 100635 to
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all 5-HT1A receptor binding sites (i.e. Rats administered daily Fluoxetine ( Prozac ) (20 mg kg(-1), i.p.) sho significantly decreased inflammatory response to subplantar carrageenan when examined on the
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5th and 14th day of Fluoxetine ( Prozac ) injection. It is suggested that Fluoxetine ( Prozac ) alone or co-administered with either imipramine or melatonin would be of benefit in the sitting of neuropathic or inflammatory pain conditions. However, two to three weeks of treatment are necessary to see the first signs
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Central serotonin receptors and chronic treatment with selective serotonin reuptake inhibitors in the rat.
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